why does the pka for h2so4 and h2so3 differ so greatly but the pka of h3po4...
Explain why Ka for H2SO4 is larger than Ka for H2SO3.
Provide a brief explanation for the following experimental observations: a) The pKa value of H2SO4 is lower than the one of H2SO3. Estimate the expected pKa values in water. b) The gas phase proton affinity of benzophenone is larger than the one of acetone. c) 4-Dimethylaminopyridine, DMAP, is a stronger Brønsted base than pyridine. d) Anhydrous phosphoric acid, H3PO4, exhibits electrical conductivity. e) Dimethylsulfoxide, DMSO, binds to AlCl3 via the oxygen atom but to AgCl via the sulfur atom
worksheet torcyclohexene" 1. Why do we use H3PO4 instead of H2SO4 as a catalyst for the synthesis of cyclohexene? (2 pts.) 2. Why do we use H3PO4 instead of HCl as a catalyst for the synthesis of cyclohexene? (3 pts.) 3. What alkeneſs) would be produced on dehydration of each of the following alcohols? If more than one product is possible, use Zaitsev's rule to predict which product would be formed in greater amounts. (9 pts.) Hz a) 2-methylcyclohexanol bi...
Please Explain: 7) a. Why does an acid and its conjugate base have the same pKa and Ka values? b. Why does a base and its conjugate acid have the same pKa and Ka values? Thank You! <3
Why is the pka value of cycloheptatriene so much greater than that of cyclopentadiene? Where pka value of cycloheptatriene =36, Pka value of cyclopentadiene=17
Why do the pKa values of ionizable groups differ between free amino acids and amino acid residues in polypeptides? thanks
4. Why do prices for the same procedure differ so significantly from one facility to another? How are health care costs hidden from consumers? 5. What role does medicalization play in rising health care costs?
4. How and why does short-run equilibrium differ in competitive and monopolistic markets? Be as specific as possible. (10) b. Profits in competitive markets and in monopoly markets have different causes and serve different purposes. Discuss. (5)
Phosphodiesterase inhibitors increase signaling through PKA. How does this work? Why does the presence of an inhibitor increase signaling and what does this say about background levels of signaling in this pathway?
Why does the MCe differ from the wage in a monopsony market?