Homework Answers
Option D is correct.
Neurotransmitter causing opening of sodium channels is excitatory in nature and cause depolarization of post synaptic neuron. ( Excitatory post synaptic potential).
While neurotransmitter causing opening of chloride ions is inhibitory in nature and cause hyperpolarization of post synaptic neuron. (Inhibitory post synaptic potential)
So according to this option a and b gets incorrect.
When both neurotransmitter work simultaneously there will be less depolarization in neuron B as compared to the condition when only neuron A sending signals.
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Conotoxin is produced by marine cone snails. Among its effects is to block voltage-gated Ca2+ channels...
Conotoxin is produced by marine cone snails. Among its effects is to block voltage-gated Ca2+ channels in neurons. A. What anatomical part of a neuron would be affected by conotoxin? B. How would the neuron’s action potential be affected by conotoxin? Explain, using at least TWO of the following terms: threshold, depolarization, repolarization, hyperpolarization, summation, IPSP, EPSP, exocytosis C. If conotoxin affected a somatic motor neuron, would this toxin cause muscle weakness or increased muscle tension? Explain why. 2.
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Fifteen different presynaptic neurons synapse on a single postsynaptic neuron. The postsynaptic neuron has a RMP of -70 mV. At the trigger zone, 14 of the presynaptic neurons produce excitatory postsynaptic potentials of 2 mV each, and the other one produces an inhibitory postsynaptic potential of 9 mV. The threshold for the postsynaptic neuron is -50 mV. Will action potentials be produced in the postsynaptic neuron? Is this an example of temporal summation or spatial summation? Explain your answers.
CNCORA 6 of 10 > Action potentials in neurons involve opening and closing of voltage-gated Nat and K ion channels. Place the events of an action potential in order, starting and ending with a cell at its resting membrane potential. Resting state Return to resting state Answer Bank A graded potential brings the membrane to threshold potential. Fast Na+ and slow K* channels are activated. Nat rushes into the cell, causing membrane depolarization. K channels close slowly, resulting in hyperpolarization....
Cell # 1 forms an excitatory synapse onto cell # 2 , when cell #1 fires a single action potential it releases glutamate onto the membrane of cell #2 resulting in a 5 millivolt depolarization, a 5 mV excitatory postsynaptic potential, when cell #1 fires three action potentials in rapid succession this causes a 15 mV depolarization in cell #2 resulting in cell #2 reaching threshold and firing its own action potential. Cell #1 Cell #2 +40 Vm IN Cell...
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Multiple Choice Which of the following statements about action potentials is incorrect? During an action potential, the efflux of K ions causes the inside of cells to become more positive (depolarized). B) When critical threshold is reached an action potential is triggered C) Voltage gated channels are required to reach critical threshold D) Sodium ions diffuse across the membrane to cause depolarization. E) K ions continue to efflux after resting membrane is reached to cause repolarization
Question 4 2 pts During and action potential, sodium (Na+) rushes into the cell causing of the cell. Then sodium channels close and potassium (K+) rushes out of the cell, causing of the cell. However, the potassium overshoots and causes Question 5 2 pts During the refractory period, the pumps Na+ ions out of the cell and K+ions into the cell, re-establishing the resting membrane potential and concentration gradient. Serotonin Acetylcholine Glutamate Endorphins Barbiturates GABA Dopamine Cocaine Sodium Potassium Calcium...
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