Question

1) Explain these symptoms of cyanide poisoning

a) Rapid breathing
b) Rapid heart rate
c) Weakness

2) During ETC, the hydrogen carriers are (reduced, oxidized, or synthesized)?

3) During ETC, oxygen is (reduced, oxidized, or synthesized) into water?

4) What are the reactants and the number of them of ETC during aerobic cellular respiration?

Answer 1

Answer :

1) Cyanide inhibits cytochrome oxidase and thus prevents cellular oxygen utilisation inhibiting electron transport chain (ETC) .This creates functional tissue hypoxia leading to metabolic acidosis .

Tissue hypoxia and metabolic acidosis lead to tachypnea (increased respiratory rate ) and tachycardia ( increased heart rate ) in an urge to compensate for metabolic needs of the body.

Since ETC is inhibited hence ATP production is hampered leading to weakness ( ATP are energy packets) .

2) During ETC hydrogen carriers like NADH are oxidised

3) Oxygen acts as final electron acceptor to produce water, thus oxygen is reduced.

4) NADH, FADH2 are reactants. Usually 10 NADH and 2 FADH2 enter ETC per molecule of glucose utilised by aerobic respiration.

Electron Transport Chain a Nath of electron NADHIH movement, NADA NABic Complex I Fabre Complex I) FADT C Complex TV 1 implex II Leyte 10₂ +24+ Complex Is NADH dehydrogenase Hok Complex II = Succinate coo Reductase Complex III = COB Cytochoume C Reductase Complex N = Cytochrome Oxidase.

Glycolysis of l Glucose molecule 2 NADH 2 NADH Link reaction between glycolysis - and Krebs cycle (oxidative decarboxylation of pyruvic acid) Krebs cycle - 6 NADH + 2FADH2 Per Glucose Molecule = 10 NADH + 2 FADH₂ u Enter Chain Electron in Aerobic Transport respiration