How did gene duplication and mutation produce the information for a third type of opsin protein?
Explain gene duplication in the X chromosome in terms of meiotic events in a XX (female) individual.
LWS gene came from the gene duplication and mutation of MWS gene located on X chromosome.
Gene duplication is a process in prophase I in meiosis where unequal crossing occurs.
How did gene duplication and mutation produce the information for a third type of opsin protein?...
how did the third opison gene arise?
Question 15 2 pts How did the third Opsin gene arise? MARK ALL THAT APPLY novel gene made from two non-functional gene fragments that fused Chromosomal translocation event gene duplication event that duplicated opsin 2 mutations arose to the duplicated opsin gene allowing greater range of color vision All of these
Short explain and type please What is the daf-2 gene and how did a mutation to this gene affect C elegans? Could a mutation to this gene affect humans the same way? Explain.
Hemophilia A is a genetic disorder of recessive X chromosome mutation of the Factor VIII gene that causes a blood clotting protein Factor VIII missing or deficient. If a person with XY chromosomes is affected by Hemophilia A and a person with XX chromosomes is an unaffected person, what is the probability that their male offspring might be hemophilic and what is the probability that their female offspring might be carriers of the mutated gene?
A DNA coding sequence of ATGCGTGGA(sequence for the rest of the gene)AATTAA encodes a protein chain of 200 amino acids of MET-ARG-GLY-(196 more amino acids)-ASN after splicing. A mutation occurs in which the third G is changed to a T. Using the genetic code below, determine which type of mutation this is and briefly explain how translation will be affected for this protein chain.
In a wild-type carnivorous plant, protein X is encoded by a haplosufficient gene “X”. The X protein normally homodimerizes. The X-X dimer enzymatically catalyzes a biochemical reaction that is critical for the digestion of insects that it traps. XD represents a dominant negative allele, a mutation, of gene X. What would be the predicted phenotype of a plant with the genotype X+/XD ? Which one of these pieces of information allows you to reach that conclusion? a. The plant would...
Questions The citrate (cit) operon 1. Operons are the basic unit of gene expression in prokaryotes. Explain the role of each of these operon components in prokaryotic gene expression: structural genes, promoter, repressor, and operator 2. Fully describe the E. coli cit operon as it functions in conditions without oxygen. Include promoter, repressor and operator in your description. 3. The cit operon is under negative control. What does that mean and how is this operon negatively controlled? 4. What is...
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part last part of question says: explain why this type of mutation
is your selected protein or protein complex would fix your initial
defect
3. (8 points) You observe a population are missing their DNA chromosome after di occurred to eliminate the function of an import packaging or localizing partitioning of the cell in two ve a population of dividing cells and discover that many of these cells some after division! You suspect that a mutation has mate the...
Draw a box-and-arrow (SMRF) model representing your understanding of HOW information in the lysozyme gene (LYZ) is used in a human cell to produce the lysozyme protein. model should include the following structures: amino acids, chromosome, DNA, gene, mRNA, nucleotides, protein. Make sure the wording of your model is specific to the example of lysozyme.
thumbs up for good answer! please help with my genetics, thank
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1. In the analysis of bacteriophage T4 rll locus mutants, a mixed infection experiment was eonducted. Complementation was ebierved for mutants 1 and 2, as well as for mutants 2 and 3 but mot between and 3. What can be concluded from the results? A-mutations 1 and 3 are on dferent genes B-all 3 mutations are in separate genes C-mutations 1 and 3...
Explain what classifies a mutation as “temperature‑sensitive” by comparing phenotype(s) with a wildtype allele. How is this type of phenotype explained by protein behaviour? Would the following types of mutations most likely have: severe effects, mild effects, or no effect on protein function? Briefly explain your answers. Note that there may be more than one correct answer, but you need only to give one sensible answer and explain your reasoning. [6 marks] A nonsense mutation occurring in sequences encoding amino...